D-ribose is a versatile metabolic substrate which may hasten improvement of post-inflammatory illness. Post-covid syndrome may be the result of torpid recovery after prolonged hypoperfusion. This reduces delivery of metabolic substrates needed to maintain cellular function. All tissues are underperfused when blood viscosity is elevated.
Therapeutic plasmapheresis is the therapy of choice for reducing blood viscosity and restoring normal perfusion. the increase in assorted plasma proteins and fibrinogen in blood plasma stimulated by a. Diastolic dysfunction, another common complication of coronavirus 2019, is a consequence of this hypoperfusion. Plasma Viscosity, gives a measure of the acute phase response i.e. Perfusion is inversely proportional to blood viscosity. The viscosity of blood depends on the viscosity of the plasma, in combination with the hematocrit and proteins. In the early twentieth century, Osler noted right ventricular dilation, a common complication of coronavirus 2019, in response to hyperviscosity caused by infection. 2 Blood is a suspension of cells in plasma. Coronary thrombosis occurs in association with several infectious diseases and even immunization. These increase erythrocyte aggregation which causes sludging of blood in areas of low shear in the coronary circulation, veins and microvasculature.
This hyperviscosity is caused by high concentrations of acute phase reactants, especially fibrinogen, and immunoglobins. The cardiac complications of coronavirus 2019 are most commonly caused by hyperviscosity, not viral invasion of endothelial cells or cardiomyocytes. This phenomenon is fully expressed in coronavirus 2019.
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